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Lee21 View Drop Down
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Post Options Post Options   Thanks (0) Thanks(0)   Quote Lee21 Quote  Post ReplyReply Direct Link To This Post Posted: Mar 29 2012 at 1:43pm

Even a Little Drinking May Raise Breast Cancer Risk: Study-Heavy consumption increases risk up to 50 percent, new review finds


http://health.msn.com/health-topics/addiction/even-a-little-drinking-may-raise-breast-cancer-risk-study
http://alcalc.oxfordjournals.org/content/early/2012/03/14/alcalc.ags011.abstract

For women with increased risk of BC (including us) -- the recommendation is to avoid alcohol or to consume alcohol only occasionally.
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1/30/12 DD AC-T, 6/7/12 Lumpectomy, ypT1b(0.8 cm), 7/9/12 Rads x 30
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Post Options Post Options   Thanks (0) Thanks(0)   Quote 123Donna Quote  Post ReplyReply Direct Link To This Post Posted: Mar 29 2012 at 1:49pm
Lee,

Thanks, good article.  I wonder if the study was broken out by breast cancer sub-type?  All I could find was:  Alcohol is thought to increase estrogen levels, in turn, perhaps, increasing the risk of breast cancer. Several studies have found alcohol more strongly linked to cancers known as estrogen receptor positive, which require estrogen to grow.  I remember seeing a study a while back that said it didn't have much effect on TNBC, only estrogen positive.  I think everything in moderation is good.  My onc advices no more than 2 glasses of wine or alcohol a day a couple of times a week. 

Donna
DX IDC TNBC 6/09 age 49, Stage 1,Grade 3, 1.5cm,0/5Nodes,KI-67 48%,BRCA-,6/09bi-mx, recon, T/C X4(9/09)
11/10 Recur IM node, Gem,Carb,Iniparib 12/10,MRI NED 2/11,IMRT Radsx40,CT NED11/13,MRI NED3/15

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Lee21 View Drop Down
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Post Options Post Options   Thanks (0) Thanks(0)   Quote Lee21 Quote  Post ReplyReply Direct Link To This Post Posted: Mar 29 2012 at 2:07pm
No, there is no breakout of TNBC subgroup.

The increase of estrogen by alcohol is only one potential mechanism, and alcohol could mediate BC risk through other mechanisms as detailed in the abstract from Alcohol and Alcoholism, including promotion of inflammatory processes and inhibition of anti-inflammatory pathways.

Ultimately, it's a lifestyle choice. Along with a 30g fat diet and moderate exercise.  For those of us with a tiny bit of ER positivity, I think it is a valid concern -- it is possible for the chemo to kill off tumor cells that are chemo-sensitive and not touch the chemo-insensitive (ER+) cells that could come back later.

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1/30/12 DD AC-T, 6/7/12 Lumpectomy, ypT1b(0.8 cm), 7/9/12 Rads x 30
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Post Options Post Options   Thanks (0) Thanks(0)   Quote 123Donna Quote  Post ReplyReply Direct Link To This Post Posted: Mar 30 2012 at 11:29am

I saw this article which I think coincides with your post.

Light Drinking Can Raise Breast Cancer Likelihood

The journal Alcohol and Alcoholismhas published a new review of studies that have researched the association between alcohol consumption andbreast cancer. The findings revealed that the risk of breast cancer rises by 5% for low level or moderate drinkers, i.e. women who have one drink per day, whilst the risk for those who consume three or more drinks daily (heavy consumption) is 40-50% higher. . . . .

. . . .According to the study's meta-analysis, which was based on the findings of over 100 studies, there was a modest, yet important link between light drinking and breast cancer, with a 5% higher risk to light drinkers compared with those who were abstinent. 

Seitz and La Vecchia's findings also showed that each higher level of alcohol consumption increases the risk of breast cancer. The findings revealed a highly important progressive upward trend in risk together with consistent evidence. 

The largest amount of research material was based on the relationship between high-level alcohol consumption and cancer risk, with findings suggesting that women who consume three or more drinks a day have an elevated breast cancer risk of 40-50%. . .
. . . .They discovered that a substantial amount of research indicates that alcohol consumption increased the risk of all ER+ tumors by 27% and posed a 14% risk for all ER- breast cancers in women with the highest alcohol consumption compared with those consuming the lowest level. The researchers discovered during later evaluations that their findings supported those of other studies, which demonstrated that the association between heavy alcohol consumption and ER+ breast cancers was substantially higher. 

With regard to ethanol-mediated breast cancer, the scientists note that few studies have been conducted and that there is only limited information available. They state that previous studies have observed a promotional effect of estrogens on breast tissue, and given that alcohol consumption causes elevated estrogen concentrations, there have been speculations that the carcinogenic effect of alcohol is partly mediated by estrogens. 

The team discussed various study examples in terms of estrogen's evidential role in ethanol-mediated breast cancer and possible carcinogens for the breast, and observed that several studies support the belief that alcohol is more strongly related to ER positive than to ER negative breast tumors, which highlights the pathogenic effect of estrogens in alcohol mediated breast cancer. 

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Post Options Post Options   Thanks (0) Thanks(0)   Quote krisa Quote  Post ReplyReply Direct Link To This Post Posted: Mar 30 2012 at 11:45am
I have never been a heavy drinker like many women I know. None of them have breast cancer but I did.
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Lee21 View Drop Down
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Post Options Post Options   Thanks (0) Thanks(0)   Quote Lee21 Quote  Post ReplyReply Direct Link To This Post Posted: Mar 30 2012 at 12:25pm
1)   In the review, ER- tumors were not broken out into TNBC vs Her2+ disease

2)   The risk for ER- tumors is not negligible -- slightly more than half for ER+ but not non existent

3)   There are substantially more ER+ tumors (>11,000 cases) than ER- tumors (approx. 4000 cases) - hence the statistics for ER+ tumors are more robust, and people tend to focus on the more prevalent tumors (as we all know)

4)   What the news snippet did not mention is that the alcohol - estrogen link is only ONE mechanism.  Ethanol is metabolized to acetaldehyde which has an array of effects including the formation of DNA adducts, inhibition of anti-oxidant pathways, inhibition of methyl transfer reactions. During it's conversion to acetaldehyde reaction oxygen species are generated and ROS are damaging to tissues as well as playing an important role in carcinogenesis.  The accumulation of acetaldehyde is dependent on the activity of ALDH, an enzyme that is polymorphic so certain racial groups are more likely to accumulate acetaldehyde.

5)   It's a lifestyle and personal choice. If a 14% risk (current risk assessment) is acceptable whereas 27% is not, then what the heck.  I am not trying to persuade anyone to give up their wine or liquor - just to make people aware of the potential risks.

6)   Alcohol consumption is only ONE potential risk factor.  Who gets breast cancer and who doesn't is likely to be dependent on many, many variables including genetic makeup (not just BRCA1/2, but polymorphisms in numerous genes - in signaling pathways, metabolic pathways, innate and adaptive immune pathways), diet, activity, environmental exposure....

The article in Alcohol and Alcoholism can be purchased online or the patient education department at your cancer center can print it out for you. When in doubt, always go to the primary literature and decide for yourself.  The review also cites many references.




Edited by Lee21 - Mar 30 2012 at 1:05pm
12/9/11 @59,IDC,grade3, TNBC,3cm(MRI),SLNB0,stage IIA, BRCA1 variant
1/30/12 DD AC-T, 6/7/12 Lumpectomy, ypT1b(0.8 cm), 7/9/12 Rads x 30
11/9/12, clinical trial cisplatin/rucaparib, cisplatin-only arm
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Post Options Post Options   Thanks (0) Thanks(0)   Quote Charlene Quote  Post ReplyReply Direct Link To This Post Posted: Mar 30 2012 at 1:53pm
Just another tidbit from the Nightly News this past week.  NBC cited some recent research that concluded that obesity and lack of exercise raised the risk of cancer for women especially. 
 
Krisa, I understand what you mean.  My husband had 7 siblings and only one of them never smoked, only consumed a very, very occasional alcoholic drink and was overall very health conscious.  She died of colon cancer at 58.  No easy explanations for any of this, I don't think.
Best wishes,
Charlene
DX 3/10 @59 ILC/TNBC
Stage 1, Grade 2, Multifocal; Lumpectomy/re-excision
SNB 0/4 nodes, BRCA-; Taxotere/Cytoxan X4, 30 rads
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Lee21 View Drop Down
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Post Options Post Options   Thanks (0) Thanks(0)   Quote Lee21 Quote  Post ReplyReply Direct Link To This Post Posted: Apr 04 2012 at 11:26am
AACR Press Releases
Cruciferous Vegetable Consumption Linked to Improved Breast Cancer Survival Rates
April 3, 2012

  • Intake associated with decreased mortality and recurrence rates.
  • Dose–response relationship observed.
  • Researchers recommend survivors eat more cruciferous vegetables.

CHICAGO — Eating cruciferous vegetables after breast cancer diagnosis was associated with improved survival among Chinese women, according to results presented at the AACR Annual Meeting 2012, held here March 31 - April 4.

“Breast cancer survivors can follow the general nutritional guidelines of eating vegetables daily and may consider increasing intake of cruciferous vegetables, such as greens, cabbage, cauliflower and broccoli, as part of a healthy diet,” said Sarah J. Nechuta, M.P.H., Ph.D., a postdoctoral research fellow at Vanderbilt University in Nashville, Tenn.
 
She and her colleagues investigated the role of cruciferous vegetables in breast cancer survival in the
Shanghai Breast Cancer Survival Study, a prospective study of 4,886 Chinese breast cancer survivors
diagnosed with stage 1 to stage 4 breast cancer from 2002 to 2006.

After adjusting for demographics, clinical characteristics and lifestyle factors, the researchers found
cruciferous vegetable intake during the first 36 months after breast cancer diagnosis was associated with a reduced risk for total mortality, breast cancer-specific mortality and recurrence in a dose–response pattern.

Across increasing quartiles of cruciferous vegetable consumption, risk for total mortality decreased by 27 percent to 62 percent, risk for breast cancer-specific mortality decreased by 22 percent to 62 percent, and risk for recurrence decreased by 21 percent to 35 percent.

Nechuta noted that cruciferous vegetable consumption habits differ between China and the United States and suggested this fact be considered when generalizing these results to U.S. breast cancer survivors.

Commonly consumed cruciferous vegetables in China include turnips, Chinese cabbage/bok choy and greens, while broccoli and brussels sprouts are the more commonly consumed cruciferous vegetables in the United States and other Western countries,” she said. “Second, the amount of intake among Chinese women is much higher than that of U.S. women. The level of bioactive compounds such as isothiocyanates and indoles, proposed to play a role in the anticancer effects of cruciferous vegetables, depend on both the amount and type of cruciferous vegetables consumed.”

She suggested that future studies with direct measurements of bioactive compounds such as isothiocyanates and host factors that influence the effects of these biological compounds be conducted to better understand the association of cruciferous vegetable intake with breast cancer outcomes.

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1/30/12 DD AC-T, 6/7/12 Lumpectomy, ypT1b(0.8 cm), 7/9/12 Rads x 30
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Post Options Post Options   Thanks (0) Thanks(0)   Quote krisa Quote  Post ReplyReply Direct Link To This Post Posted: Apr 04 2012 at 3:21pm
Aspirin, aspirin, aspirin! Just received my daily email from Hester Hill...compelling research on aspirin in preventing cancer or metastatic cancer. Hope to post it soon.
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Post Options Post Options   Thanks (0) Thanks(0)   Quote krisa Quote  Post ReplyReply Direct Link To This Post Posted: Apr 04 2012 at 4:46pm
TUESDAY, March 20 (HealthDay News) -- Aspirin, a popular weapon in the war against heart attacks, may also play a role in cancer prevention and treatment, three new British studies suggest.
"We have now found that after taking aspirin for three or four years there starts to be a reduction in the number of people with the spread of cancers, so it seems as well as preventing the long-term development of cancers, there is good evidence now that it is preventing the spread of cancers," said lead researcher Dr. Peter M. Rothwell, a professor of neurology at the University of Oxford and John Radcliffe Hospital in Oxford.
"Because aspirin prevents the spread of cancers, it could potentially be used as a treatment," he added.
But the research is not conclusive, and did not prove that aspirin combats cancer. So, people should not start popping aspirin in the hopes of thwarting cancer, experts said.
Previously, these investigators showed that a daily dose of aspirin taken over 10 years appeared to prevent some cancers, but the short-term benefits and the benefits for women weren't clear.
Currently, a daily low-dose aspirin is recommended for people who have had a heart attack or stroke to prevent another. "It may well be that taking aspirin to prevent cancer becomes the main reason for taking it," Rothwell said.
Aspirin may work against cancer by inhibiting platelets, which promote clotting and also help cancer cells spread, he said.
The papers were published March 21 in The Lancet and The Lancet Oncology.
In one study, Rothwell's team analyzed data from 51 clinical trials comparing aspirin with no aspirin in preventing
heart attacks.
Overall, daily low-dose aspirin reduced the risk of dying from cancer 15 percent. Taking aspirin five years or more reduced the risk 37 percent, and over three years, the risk reduction was about 25 percent for both men and women, the researchers noted.
In addition, aspirin was associated with a 12 percent reduction in deaths from non-cardiovascular causes, they found.
In another study, Rothwell's team looked at the effect of aspirin on slowing the spread of cancer, or metastasis.
Their data came from five clinical trials that also looked at daily low-dose aspirin (75 milligrams or more) and heart attack and stroke prevention. The researchers zeroed in on patients who developed cancer.
Over more than six years of follow-up, low-dose aspirin reduced the risk of distant metastasis by 36 percent, compared with cancer patients receiving a placebo, they found.
Moreover, aspirin reduced the risk of metastasis in solid tumors, such as colon, lung and prostate cancer, by 46 percent and by 18 percent for cancers of the bladder and kidney.
It also reduced the risk of diagnosing a cancer that had already spread by 31 percent. For those who continued to
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Post Options Post Options   Thanks (0) Thanks(0)   Quote krisa Quote  Post ReplyReply Direct Link To This Post Posted: Apr 04 2012 at 4:48pm
Take aspirin after a cancer diagnosis, the risk of metastasis was cut by 69 percent, the researchers calculated.
Aspirin also reduced the risk of dying from cancer by about half. These risk reductions remained after taking into account age and sex, the researchers said.
In a third study, Rothwell's group looked at the effect of aspirin on metastases by analyzing observational studies rather than clinical trials.
These studies revealed a 38 percent reduction in colon cancer, which matched well with the risk reduction seen in clinical trials, they said. There were similar findings for esophageal, gastric, biliary and breast cancer, they added.
While the study is attention-getting, not everyone agrees with the overall conclusions.
Among them is Nancy R. Cook, an associate biostatistician at Brigham and Women's Hospital and Harvard Medical School in Boston and co-author of an accompanying journal editorial. She pointed out that these studies only dealt with trials where aspirin was given daily, whereas two large trials in which aspirin was given every other day found no connection with cancer prevention.
"Aspirin seems to work for people who have had cardiovascular disease. Perhaps in the long-term it will turn out to be protective for cancer, but we need to verify that and get more information," Cook said.
And, aspirin is not benign, Cook said, pointing out risks for bleeding and other gastrointestinal problems.
People should not start taking aspirin hoping to preventing cancer, Cook said. "Most of the studies show that the effect doesn't accrue until after 10 years," she noted.
Eric Jacobs, strategic director of pharmacoepidemiology for the American Cancer Society, said that "this study provides important new evidence that long-term daily aspirin, even at low doses, may lower risk of developing cancer."
However, any decision about treatment should be made on an individual basis in consultation with a doctor, he said.
"Because these results are new," Jacobs added, "it will take time for the broader scientific community to evaluate the data in the context of existing knowledge and to consider whether the clinical guidelines should be changed."
More information
For more on cancer, visit the American Cancer Society.


Edited by krisa - Apr 04 2012 at 4:49pm
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Post Options Post Options   Thanks (0) Thanks(0)   Quote Grateful for today Quote  Post ReplyReply Direct Link To This Post Posted: Jul 25 2012 at 10:51pm
Hi,

"bumping up" for members who have joined since April.

Lots of things to consider and interesting posts on this thread/forum topic.
Some of the things have not been proven in human clinical trials.
Many things won't hurt and may help......so worth knowing about.

With caring and positive thoughts,
Grateful for today.........Judy
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Post Options Post Options   Thanks (0) Thanks(0)   Quote Lee21 Quote  Post ReplyReply Direct Link To This Post Posted: Dec 02 2012 at 3:42pm
Pomegranate juice anyone?

Breast Cancer Res Treat. 2012 Oct 12. [Epub ahead of print]

Pomegranate juice and specific components inhibit cell and molecular processes critical for metastasis of breast cancer.

Source

Department of Cell Biology and Neuroscience, University of California Riverside, BSB Room 2217, 900 University Avenue, Riverside, CA, 92521, USA.

Abstract

Breast cancer is the most common cancer and the second leading cause of cancer death and morbidity among women in the western world. Pomegranate juice (PJ) and three of its specific components have been shown to inhibit processes involved in prostate cancer metastasis. If this also proves to be true for breast cancer, these natural treatments will be promising agents against breast cancer that can serve as potentially effective and nontoxic alternatives or adjuncts to the use of conventional selective estrogen receptor modulators for breast cancer prevention and treatment. To test this possibility, we have used two breast cancer cell lines, MDA-MB-231 cells (ER(-)) and MCF7 (ER(+)), and the non-neoplastic cell line MCF10A. We show that, in addition to inhibiting growth of the breast cancer cells, PJ or a combination of its components luteolin (L) + ellagic acid (E) + punicic acid (P) increase cancer cell adhesion and decrease cancer cell migration but do not affect normal cells. These treatments also inhibit chemotaxis of the cancer cells to SDF1α, a chemokine that attracts breast cancer cells to the bone. We hypothesized that PJ and L + E + P stimulate expression of genes that increase adhesion and inhibit genes that stimulate cell migration and inhibit chemotaxis to SDF1α. Using qPCR, we confirmed these proposed effects on gene expression and in addition we found that a gene important in epithelial-to-meshenchymal transitions is decreased. We also found that pro-inflammatory cytokines/chemokines are significantly reduced by these treatments, thereby having the potential to decrease inflammation and its impact on cancer progression. Discovery that PJ and L + E + P are inhibitory of metastatic processes in breast cancer cells in addition to prostate cancer cells indicate that they are potentially a very effective treatment to prevent cancer progression in general.

PMID:
 
23065001
 
[PubMed - as supplied by publisher]

MCF-7 cell line is ER positive
MDA-m?-231 cell line is ER negative (TN)
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1/30/12 DD AC-T, 6/7/12 Lumpectomy, ypT1b(0.8 cm), 7/9/12 Rads x 30
11/9/12, clinical trial cisplatin/rucaparib, cisplatin-only arm
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Post Options Post Options   Thanks (0) Thanks(0)   Quote Lee21 Quote  Post ReplyReply Direct Link To This Post Posted: Dec 02 2012 at 3:45pm
Bump since above post did not update thread
12/9/11 @59,IDC,grade3, TNBC,3cm(MRI),SLNB0,stage IIA, BRCA1 variant
1/30/12 DD AC-T, 6/7/12 Lumpectomy, ypT1b(0.8 cm), 7/9/12 Rads x 30
11/9/12, clinical trial cisplatin/rucaparib, cisplatin-only arm
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Post Options Post Options   Thanks (0) Thanks(0)   Quote beck Quote  Post ReplyReply Direct Link To This Post Posted: Jan 17 2013 at 2:08pm
i try to walk alot join the y but that didnt work for me trying to cut down on sweets and eat veg. and fruit, its hard to teach a dog new tricks
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Post Options Post Options   Thanks (0) Thanks(0)   Quote Grateful for today Quote  Post ReplyReply Direct Link To This Post Posted: Apr 19 2013 at 11:23pm
Hi,

On another thread today, ddspain posted:

Originally posted by ddspain ddspain wrote:

Hi !
There has been a suggestion that we should think about lifestyle changes .

I agree and wonder.....

what lifestyle changes have been the most rewarding or effective for all of you ?
I appreciate your responses.


Thank you,
D.


Realize this thread has lots of posts on Lifestyle changes.
Any thoughts on the "most rewarding and most effective" lifestyle changes?


Grateful for today............Judy
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Post Options Post Options   Thanks (0) Thanks(0)   Quote denise07 Quote  Post ReplyReply Direct Link To This Post Posted: Apr 19 2013 at 11:59pm
My opion only it is all in our genes. Weight factor never a problem for me,veggie lover yes I am broccoli sure lover I am 48 so consumed that for 48 years of my life mom says even with baby food if you look at things with some reason, why? do some people smoke and drink heavaly through out there lives have not one health problem not that I would wish that and then we have people who watch every thing they consume end up with cancer?Genetic? I pray everyday for a end for this awful disease.This is definatley one crap shoot and I pray they figure it out soon.
My opionion only...
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Post Options Post Options   Thanks (0) Thanks(0)   Quote Annie Quote  Post ReplyReply Direct Link To This Post Posted: Apr 20 2013 at 1:27pm


    Hi All,   It can be akin to running to and fro when we receive information that this or that causes or prevents Cancer or its recurrence...While it is very good and possibly quite helpful to adopt some of these supposed to be investigated avenues we need to really remember so that we will not be driven to despair that the cause of Cancer still is shrouded in Mystery. All we can do is the best we can...take care all...Love, Annie
Annie TNBC Stage IIA Gr 3 1cm lesion 2/5 lymph nodes+ lumpectomy,FEC & D 30Rads finished(08/2009) BRCA- Chronic Cellulitis due to Radiation-- L.Mastectomy Jan 2012
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Post Options Post Options   Thanks (0) Thanks(0)   Quote CrunchyGirl Quote  Post ReplyReply Direct Link To This Post Posted: May 26 2014 at 12:53am
I have been wondering what changes all of you NED have made as well. I am just finishing chemo and this is what I'm going to do now that I'm getting my body back from the drugs...

1.Wheatgrass juicing everyday
2. Cannabis oil high CBD each night
3. Frankincense oil internally (lots of research coming out , so why not?)
4. More veggies, more exercise.
5. NO more soy, GMOs, too much alcohol, white sugar (hardest one)
3cm in left 31yrs 4 Red devil, 12 taxol, neg nodes, surgery July 2014. Tumors shrunk by 1cm all negative. Now cancer Free!
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Post Options Post Options   Thanks (0) Thanks(0)   Quote 123Donna Quote  Post ReplyReply Direct Link To This Post Posted: May 26 2014 at 11:15pm
It's common knowledge many women gain weight after going through treatment.  I don't know how helpful this article is or if it really is beneficial.  (The study was done on mice.)  All I know is no one should feel guilty about weight gain caused by treatments.  

Women With Breast Cancer May Benefit From Restricted Diets

DX IDC TNBC 6/09 age 49, Stage 1,Grade 3, 1.5cm,0/5Nodes,KI-67 48%,BRCA-,6/09bi-mx, recon, T/C X4(9/09)
11/10 Recur IM node, Gem,Carb,Iniparib 12/10,MRI NED 2/11,IMRT Radsx40,CT NED11/13,MRI NED3/15

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